RF2RTXHMC–Test for clubbed fingers, The Schamroth window test. Woman pushing her thumb nails together showing bright gap and passing the test. Red nail varnish.
RMATB0B3–Finger clubbing
RM2AGA7GT–. Diseases of children. Fig. 46 —The clubbed fingers of chronic heart disease..
RMCRYYJ3–PATHOLOGY OF THE NAIL, DRAWING
RMT23CMG–Archive image from page 118 of Discovery reports (1932) Discovery reports discoveryreports06inst Year: 1932 AMMOTHEIDAE i°7 Abdomen elevated at an angle of about 45' and reaching to distal end of fourth lateral process. ChelopJiore short, less than one-third of proboscis; scape short, clubbed; chela small, with fingers reduced to small blunt stumps. Palp rather longer than proboscis, with nine segments (Fig. 57 b); fourth almost twice as long as second segment. Oviger as represented in Fig. 57 a. Fig. 57. AmmotJiea stylirostns, n.sp. Holotype: rt. Oviger: ;<i5. 6. Palp: xis. c. Termina
RMA78PKP–Finger clubbing
RM2AN2B52–Diseases of children, for nurses, including infant feeding, therapeutic measures employed in childhood, treatment for emergencies, prophylaxis, hygiene, and nursing . Fig. 45.—The clubbed fingers of chronic heart disease.. Fig. 46.—Clubbing of toes in chronic heart disease. General Cyanosis.—Blueness of the surface frominsufficient oxidation of the blood. Clubbing of the fingers in chronic cases. DISEASES OF THE CIRCULATORY SYSTEM 8l PERICARDITIS This is an inflammation of the pericardium or serouscovering of the heart. It is rare in infancy, but as thechild grows older it is not uncommon. I
RMATB157–Finger clubbing secondary to lung cancer
RM2AFP78Y–. Internal medicine; a work for the practicing physician on diagnosis and treatment, with a complete Desk index. Aiiniristn of ascemiinuarch of the aorta. 678 MEDICAL DIAGNOSIS.. Fig. 360.—Unilateral clubbed fingers in aneurism of thedescending arm of the aortic arch.—Groedels case. and in older cases the A^oice may be unchanged. Pupillary derangementsare sometimes observed. Compression of the trachea and left bronchus,and tracheal tugging are very common. Dysphagia is more frequentthan in aneurisms in other portions of the aorta. The sac sometimesincludes portions of the ascending and transve
RMATB597–Ca bronchus finger clubbing and nicotine stains
RM2ANGDNK–Diseases of the chest and the principles of physical diagnosis . n-justified. Pulmonary stenosis is a rare and nearly always, congenitallesion which is associated with cyanosis, clubbed fingers, cardiac hyper-trophy and a feeble second sound. 8. The diastolic pulmonic murmur (pulmonary insufiiciency) mayresult from congenital disease, or from ulcerative endocarditis; in eithercase it is a rare lesion. It may be due to functional dilatation of the pul- 16 242 THE EXAMIXATION OF CIRCULATORY SYSTEM monarv orifice under severe strain which leads to increased blood-pressurein the lesser circulation
RM2AJ4RC6–The report of the Philadelphia baby saving show, and the proceedings of the Conference on infant hygiene . with marasmus. Colored Drawing. A hand showing clubbed fingers from congenital heart disease. DIIIFA TOUY AM) ( ATAIJXiri-: OF FXIUIUTS 99 (ol.nr.l I )ra in^s of (liilcircri. Illiisl raliiii^- llic lollowiiiff tiilxTciilmis lesions:liil)ircul()sis of till dhow,riihcrciilous hip diseaso.riil)onnlosis of the knee.Tiilierciilosis of tlie ankle-joint.Earl liilKTciHosis of the spine.Iuhereulosis of the sjjiTial roluniii.rnherenloiis peritonitis,rnherctilons inflammation of the hiain. ( (ill
RM2CDATJF–. Physical diagnosis . Fig. 50.—Atrophic Arthritb.. Fig. 51.—Clubbed Fingers. 5. Acromegalia produces general enlargement of the bones andother tissues of the hands and feet. 6. Pulmonary Osteo-arthropathy.—Any long-standing disease of THE HANDS 53 the heart, lungs, or pleura may be followed by this peculiar hyper-trophic change in all the tissues of the extremities. Mild formsproduce clubbed fingers, a bulbous enlargement of the finger-tips
RM2CDAT7D–. Physical diagnosis . Fig. 51.—Clubbed Fingers. 5. Acromegalia produces general enlargement of the bones andother tissues of the hands and feet. 6. Pulmonary Osteo-arthropathy.—Any long-standing disease of THE HANDS 53 the heart, lungs, or pleura may be followed by this peculiar hyper-trophic change in all the tissues of the extremities. Mild formsproduce clubbed fingers, a bulbous enlargement of the finger-tips. Fig. 52.—Clubbed Fingers.
RM2CRT4NA–. A practical treatise on medical diagnosis for students and physicians . W Pseudomuscular atrophy. Claw-hand. (Gray.) so-called strumous persons, and the effeminate hand of one who is inclinedto tuberculosis, present sharp contrasts. Then, too, the occupation hand indicates in a general way the disease the patient is liable to—nonemore striking than the hand of the miner, the blue-black dottings of THE HANDS.- 197 which clearly indicate the possibility of anthracosis. Finally, we notethe broad hand and clubbed fingers that are seen in congenital heartdisease. The withered hand of age and wast
RM2CGGT2M–. Physical diagnosis . Clubbed Fingers. sijncope ( dead fingers) following exposures to slight cold or emo-tional strain. The fingers become white and cold. The conditionusually passes off in an hour or two. From similar causes we mayhave: B. Local asphyxia (chilblains), producing congestion andswelling with or without pain and stiffness and with heat or coldnessof the part. C. Local or symmetrical gangrene. If local asphyxiapersists, gangrene results.4 50 PHYSICAL DIAGNOSIS. 11. Morvans Disease.—As a part of syringomyelia multiplearthropathies (atrophic arthritis) and painless felons may deve
RM2CGGTPD–. Physical diagnosis . Fig. 28.—Tetany. (Masland.) THE HANDS. 47 5. Acromegalia produces general enlargement of the bones andother tissues of the hands and feet. 6. Pulmonary Osteo-arthropathy.—Any long-standing diseaseof the heart, lungs, or pleura may be followed by this peculiar hy-pertrophic change in all the tissues of the extremities. Mild formsproduce clubbed fingers, a bulbous enlargement of the finger-tipswith double curvation of the nails, lateral and antero-posterior (see. FIG. 29.—Atrophic Arthritis with Flipper Hand. Fig. 31). In severer forms the bones of the hand and wrist are a
RM2CDAT04–. Physical diagnosis . Fig. 52.—Clubbed Fingers.. Fig. 53.—Raynauds Disease. with double curvation of the nails, lateral and antero-posterior1 (seeFig. 51). In severer forms the bones of the hand and wrist are alsoconsiderably enlarged (see Figs. 39 and 40). 1 Clubbed fingers are occasionally seen in a variety of other diseases: e.g., hepaticabscess, nephritis; and even in apparently healthy persons. 54 PHYSICAL DIAGNOSIS 7. Heberdens nodes, later described under the head of hypertrophicarthritis, are here pictured (Fig. 54). The distinction from gout hasalready been referred to (page 472). 8.
RM2CDARWR–. Physical diagnosis . Fig. 53.—Raynauds Disease. with double curvation of the nails, lateral and antero-posterior1 (seeFig. 51). In severer forms the bones of the hand and wrist are alsoconsiderably enlarged (see Figs. 39 and 40). 1 Clubbed fingers are occasionally seen in a variety of other diseases: e.g., hepaticabscess, nephritis; and even in apparently healthy persons. 54 PHYSICAL DIAGNOSIS 7. Heberdens nodes, later described under the head of hypertrophicarthritis, are here pictured (Fig. 54). The distinction from gout hasalready been referred to (page 472). 8. Atrophic arthritis (Fig. 4
RM2AWT7F5–Nervous and mental diseases . diagnosis can rarely presentdifficulty if the disease has attained any consider-able degree of development. Myxedema shows no osseous deformities, and the thickening of the soft parts is waxy andboggy. Pulmonary ostco-arthropathy presents joint-changes and a chronicpulmonary process. The clubbed enlargement of the fingers is largelyconfined to the terminal phalanges, over which the hypertrophic nailshang like parrot-beaks. In hyperostosis cranii the hands, feet, andmandible are not affected. Pagets disease, or osteitis deformans, spares theface, but affects the sk
RM2AWYXJM–Diseases of children for nurses . Fig. 6.—Genu valgum. Female childfive years old (Napier). Fig. -Genu varum from rachitis(Xapier). Scurvy is seen in artificially fed infants. Rickets and marasmus are typical nutritional diseasesseen only in childhood. Orthopedics.—Many children have deformities due torickets, spinal curvatures, congenital dislocation of thehips, and contractions of the tendons. These deformitiesare overcome by surgical treatment. 24 DISEASES OF CHILDREN FOR NURSES Various terms are given to the different deformities:Clubbed hands; webbed fingers; congenital dislocation ofthe
RM2AGAJ3T–. Diseases of children. Fig. 6.—Genu valgum. Bemale childfive years old (Napier). Fig. 7.—Genu varum from rachitis(Napier). Scurvy is seen in artificially fed infants. Rickets and marasmus are typical nutritional diseasesseen only in childhood. Orthopedics.—Many children have deformities due torickets, spinal curvatures, congenital dislocation of thehips, and contractions of the tendons. These deformitiesare overcome by surgical treatment. 28 DISEASES OF CHILDREN FOR NURSES Various terms are given to the different deformities:Clubbed hands; webbed fingers; congenital dislocation ofthe hips; kn
RM2AWYWYK–Diseases of children for nurses . Fig. 6.—Genu valgum. Female childfive years old (Napier). Fig. -Genu varum from rachitis(Xapier). Scurvy is seen in artificially fed infants. Rickets and marasmus are typical nutritional diseasesseen only in childhood. Orthopedics.—Many children have deformities due torickets, spinal curvatures, congenital dislocation of thehips, and contractions of the tendons. These deformitiesare overcome by surgical treatment. 24 DISEASES OF CHILDREN FOR NURSES Various terms are given to the different deformities:Clubbed hands; webbed fingers; congenital dislocation ofthe
RM2ANGT48–Peroral endoscopy and laryngeal surgery . yearspreviously the child, then six years old, aspirated a nail, followed byparoxysms of coughing and gradually failing health. Sputum examina-tion negative as to tubercle bacilli. Mixed pus cocci and saprophyteswere present. Dr. H. T. Price reported the results of his physical e.x-amination as follows: Child fairly well developed, rather languid, color good, head large,fingers markedly clubbed (Fig. 1!)3), toes not so large, slight cough atintervals of half to two minutes. Breath very offensive after cough- FORKICN liODIK.S I r,R(t.CUI FOR PROLONGED
RM2CEPWFG–. The diseases of infancy and childhood. lesion,although cyanosis alone is not to be depended upon. Another frequent symptom is the enlargement of the terminalphalanges known as clubbed or drum-stick fingers (Fig. 81) and toes.This almost invariably accompanies cyanosis, and is generally propor-tionate to it. The enlargement, which usually involves all the phalanges,is probably due to venous obstruction. Occasionally there are seen dysp- 584 DISEASES OF THE CIRCULATORY SYSTEM nea, edema of the face or lower extremities, dropsy of the serous cavities,and hemorrhages, particularly hemoptysis and
RMRCDYHR–. Discovery reports. Discovery (Ship); Scientific expeditions; Ocean; Antarctica; Falkland Islands. AMMOTHEIDAE i°7 Abdomen elevated at an angle of about 45" and reaching to distal end of fourth lateral process. ChelopJiore short, less than one-third of proboscis; scape short, clubbed; chela small, with fingers reduced to small blunt stumps. Palp rather longer than proboscis, with nine segments (Fig. 57 b); fourth almost twice as long as second segment. Oviger as represented in Fig. 57 a.. Fig. 57. AmmotJiea stylirostns, n.sp. Holotype: rt. Oviger: ;<i5. 6. Palp: xis. c. Terminal segme
Download Confirmation
Please complete the form below. The information provided will be included in your download confirmation